What is a heart attack?A heart attack (also known under the name of myocardial infarction) is the death of the muscle of heart of the sudden filling of a coronary artery by a clot of blood. The coronary arteries are blood vessels which provide the muscle of heart blood and oxygen. The filling of a coronary artery deprives the muscle of heart of blood and oxygen, causing damage with the muscle of heart. The damage with the muscle of heart causes the pain and the pressure of trunk. If the flow of blood is not reconstituted within 20 to 40 minutes, the irreversible death of the muscle of heart will start to occur. The muscle continues to die during six to eight hours when that the heart attack is usually “achieve. ” The dead muscle of heart is replaced by fabric of scar. Roughly a million Americans suffer a heart attack every year. Four hundred thousands of them stamps because of their heart attack. To click here to look at the interactive photographs of the hearts which suffered a heart attack.
How a heart attack is treated?
The treatment of the heart attacks include: drugs of Anti-plate to prevent the formation of the clots of blood in the arteries. Drugs of anticoagulant to prevent the growth of the clots of blood in the arteries. The coronary angiography with one or the other the coronary angioplastie transluminale percutaneous (PTCA) with or without stenting to open blocked the coronary arteriesthe drugs of Clot-dissolution to be opened blocked arteries. Additional oxygen to increase the supply oxygen with the muscle of the heart. Drugs to decrease the need for oxygen by the muscle of the heart. Drugs to prevent abnormal rates/rhythms of heart. The primary education goal of the treatment is to open the blocked artery quickly and to reconstitute the flow of blood to the muscle of heart, a D-perfusion called of process. Once the artery is opened, the damage with the muscle of heart ceases, and the patient becomes free pain. By to the minimum reducing the extent of the damage of muscle of heart, the D-perfusion early preserves the function of pumping of the heart. The optimal advantage is obtained if the D-perfusion can be established in the four first at six hours of a heart attack. To delay by establishing the D-perfusion can have like consequence of the more widespread damage to the muscle of heart and a greater reduction in the capacity of the heart to pump blood. The patients presenting the hearts which cannot pump sufficient blood develop the cardiac arrest, the capacity decreased to be exerted, and the abnormal rates/rhythms of heart. Thus, the quantity of healthy muscle of heart remaining after a heart attack is the determining cause most important of the future quality of the life and longevity.
